LA CONSULTA SEMANAL

ENERO 2001

CONSULTA

Arch Intern Med 2000 Sep 11;160(16):2429-36
New guidelines for potassium replacement in clinical practice: A contemporary review by the national council on potassium in clinical practice.
Cohn JN, Kowey PR, Whelton PK, Prisant LM
Cardiovascular Division, MMC 508, University of Minnesota, 420 Delaware St SE, Minneapolis, MN 55455, USA.
[Medline record in process]
This article is the result of a meeting of the National Council on Potassium in Clinical Practice. The Council, a multidisciplinary group comprising specialists in cardiology, hypertension, epidemiology, pharmacy, and compliance, was formed to examine the critical role of potassium in clinical practice. The goal of the Council was to assess the role of potassium in terms of current medical practice and future clinical applications. The primary outcome of the meeting was the development of guidelines for potassium replacement therapy. These guidelines represent a consensus of the Council members and are intended to provide a general approach to the prevention and treatment of hypokalemia. Arch Intern Med. 2000;160:2429-2436


N Engl J Med 1999 Jan 14;340(2):154-5; discussion 155
Published erratum appears in N Engl J Med 1999 Feb 25;340(8):663
Treatment of hypokalemia.
Agarwal A, Wingo CS
Publication Types:
  Comment
  Letter
Comments:
  Comment on: N Engl J Med 1998 Aug 13;339(7):451-8


N Engl J Med 1999 Jan 14;340(2):155
Treatment of hypokalemia.
Robertson JI
Publication Types:
  Comment
  Letter
Comments:
  Comment on: N Engl J Med 1998 Aug 13;339(7):451-8

Rev Prat 1998 Oct 1;48(15):1697-703
[Hypokalemia. Etiology, physiopathology, diagnosis, treatment].
[Article in French]
Dussaule JC, Tharaux PL
Service de Physiologie, CHU Saint-Antoine, hopital Saint-Antoine, Paris.
Publication Types:
  Review
  Review, tutorial


N Engl J Med 1998 Aug 13;339(7):451-8
Hypokalemia.
Gennari FJ
Department of Medicine, University of Vermont College of Medicine, Burlington
05401, USA.
Publication Types:
  Review
  Review, tutorial
Comments:
  Comment in: N Engl J Med 1999 Jan 14;340(2):154-5; discussion 155
  Comment in: N Engl J Med 1999 Jan 14;340(2):155


Lancet 1998 Jul 11;352(9122):135-40 [Texto completo]
Potassium.
Halperin ML, Kamel KS
Division of Nephrology, St Michael's Hospital, University of Toronto, Ontario, Canada. mitchell.halperin@utoronto.ca
In a logical, stepwise approach to patients presenting with hypokalaemia or hyperkalaemia the clinician must first recognise circumstances in which the dyskalaemia represents a clinical emergency because therapy then takes precedence over diagnosis. If a dyskalaemia has been present for a long time, there is an abnormal renal handling of K+. The next step to analyse is the rate of excretion of K+ and, if necessary, its two components (urine flow rate and K+ concentration in the cortical collecting duct [CCD]) analysed independently. If the K+ concentration in the CCD is not in the expected range, its basis should be defined at the ion-channel level in the CCD from clinical information that can be used to deduce the relative rates of reabsorption of Na+ and Cl- in the CCD. This analysis provides the basis for diagnosis and may indicate where non-emergency therapy should then be directed.
Publication Types:
  Review
  Review, tutorial


Med Clin North Am 1997 May;81(3):611-39
Hypokalemia and hyperkalemia.
Mandal AK
Section of Nephrology, Department of Veterans Affairs Medical Center, Dayton, Ohio, USA.
This article discusses the causes and nature of hypokalemia and hyperkalemia. Diagnosis, testing, drug administration, and general management are outlined in detail.
Publication Types:
  Review
  Review, tutorial


BMJ 1996;312:1652-1653 (29 June)
General practice
High ambient temperature: a spurious cause of hypokalaemia
P W Masters, senior registrar,a N Lawson, consultant biochemist,a C B Marenah, consultant chemical pathologist,a L J Maile, general practitioner b


Hosp Pract (Off Ed) 1995 Jul 15;30(7):67-71, 74-5, 79
Unraveling the causes of hypertension and hypokalemia.
Steigerwalt SP
Division of Nephrology and Hypertension, Henry Ford Hospital, Detroit, USA.
Coexistence of the two conditions may be coincidental, so the first step is to rule out nonrenal causes. Overall, the two most common causes are diuretic therapy and primary aldosteronism. New clinical insights regarding three other conditions--glucocorticoid-remediable aldosteronism, apparent mineralocorticoid excess, and deoxycorticosterone hypersecretion syndrome--are also discussed.
Publication Types:
  Review
  Review, tutorial


Hosp Pract (Off Ed) 1988 Mar 30;23(3A):55-64, 66, 70
Hypokalemia. Common and uncommon causes.
Stein JH
Department of Medicine, University of Texas Medical School, San Antonio.